Role of endogenous and exogenous nitric oxide, carbon monoxide and hydrogen sulfide in HCT116 colon cancer cell proliferation-Reference-Cited by-同舟云学术

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Role of endogenous and exogenous nitric oxide, carbon monoxide and hydrogen sulfide in HCT116 colon cancer cell proliferation

Published:2018-03 Issue: Volume:149 Page:186-204
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Oláh Gabor,Módis Katalin,Törö Gabor,Hellmich Mark R.,Szczesny Bartosz,Szabo Csaba

Funder

Cancer Prevention and Research Institute of Texas

University of Exeter

National Institutes of Health

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference54 articles.

1. Gasotransmitters in cancer: from pathophysiology to experimental therapy;Szabo;Nat. Rev. Drug Discov.,2016

2. Inhibition of hydrogen sulfide biosynthesis sensitizes lung adenocarcinoma to chemotherapeutic drugs by inhibiting mitochondrial DNA repair and suppressing cellular bioenergetics;Szczesny;Sci. Rep.,2016

3. Intramitochondrial hydrogen sulfide production by 3-mercaptopyruvate sulfurtransferase maintains mitochondrial electron flow and supports cellular bioenergetics;Módis;FASEB J.,2013

4. Cystathionine-β-synthase inhibition for colon cancer: enhancement of the efficacy of aminooxyacetic acid via the prodrug approach;Chao;Mol. Med.,2016

5. Selective pharmacological inhibition of distinct nitric oxide synthase isoforms;Southan;Biochem. Pharmacol.,1996

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