β-estradiol adjusts intestinal function via ERβ and GPR30 mediated PI3K/AKT signaling activation to alleviate postmenopausal dyslipidemia-Reference-Cited by-同舟云学术

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β-estradiol adjusts intestinal function via ERβ and GPR30 mediated PI3K/AKT signaling activation to alleviate postmenopausal dyslipidemia

Published:2020-10 Issue: Volume:180 Page:114134
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Meng Qinghai,Li Jun,Chao Ying,Bi Yunhui,Zhang Weiwei,Zhang Yuhan,Ji Tingting,Fu Yu,Chen Qi,Zhang Qichun,Li Yu,Bian Huimin

Funder

National Natural Science Foundation of China

Jiangsu Key Laboratory for Pharmacology and Safety Evaluation of Chinese Materia Medica

Priority Academic Program Development of Jiangsu Higher Education Institutions

Jiangsu Province

Qing Lan Project

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference42 articles.

1. Association of age at onset of menopause and time since onset of menopause with cardiovascular outcomes, intermediate vascular traits, and all-cause mortality: a systematic review and meta-analysis;Muka;JAMA Cardiol.,2016

2. Estrogen deprivation, rather than age, is responsible for the poor lipid profile and carbohydrate metabolism in women;Senoz;Maturitas,1996

3. Endogenous estrogens, estrogen metabolites, and breast cancer risk in postmenopausal Chinese women;Moore;J. Natl Cancer Inst.,2016

4. Oestrogen Receptor-alpha binds the FOXP3 promoter and modulates regulatory T-cell function in human cervical cancer;Adurthi;Sci. Rep.,2017

5. Estrogen signaling: An emanating therapeutic target for breast cancer treatment;Saha;Eur. J. Med. Chem.,2019

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4. Oestrogens, adipose tissues and environmental exposures influence obesity and diabetes across the lifecycle;Proceedings of the Nutrition Society;2024-02-02

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