Response of glutamic acid decarboxylase to glucose but not arginine in islets
Author:
Publisher
Elsevier BV
Subject
General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine
Reference18 articles.
1. Antibodies to a 64,000 Mr human islet cell antigen precede the clinical onset of insulin-dependent diabetes.
2. Persistence of serum antibodies to 64,000-Mr islet cell protein after onset of type I diabetes
3. Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase
4. Autoantibodies to GABA-ergic Neurons and Pancreatic Beta Cells in Stiff-Man Syndrome
5. Autoimmunity to two forms of glutamate decarboxylase in insulin-dependent diabetes mellitus.
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1. Modulation of Glucocorticoid-Induced GAD Expression in Pancreatic β-Cells by Transcriptional Activation of the GAD67 Promoter and Its Possible Effect on the Development of Diabetes;Diabetes;2002-09-01
2. Streptozotocin Upregulates GAD67 Expression in MIN6N8a Mouse Beta Cells;Journal of Autoimmunity;2002-08
3. Heterogeneity in glutamic acid decarboxylase expression among single rat pancreatic beta cells;Diabetologia;1999-08-19
4. The TATA-less rat GAD65 promoter can be activated by Sp1 through non-consensus elements;Gene;1999-08
5. Physiological Aspects of GAD-GABA System in the Pancreatic Islet;GABA: Receptors, Transporters and Metabolism;1996
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