Physiological Aspects of GAD-GABA System in the Pancreatic Islet
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Publisher
Birkhäuser Basel
Link
http://link.springer.com/content/pdf/10.1007/978-3-0348-8990-2_2.pdf
Reference14 articles.
1. Okada Y, Taniguchi H, Shimada C. High concentration of GABA and high glutamate decarboxylase activity in rat pancreatic islets and human insulinoma. Science 1976; 194: 620–622.
2. Taniguchi H, Okada Y, Seguchi H, Shimada C, Seki M, Tsutou A, Baba S. High concentration of gamma-aminobutyric acid in pancreatic beta cells. Diabetes 1979; 28: 629–633.
3. Christgau S, Schierbeck H, Aanstoot H-J, Aagaard L, Begley K, Kofod H, Hejnaes K, Baekkeskov S. Pancreatic 0 cells express two autoantigenic forms of glutamic acid decarboxylase, a 65-kDa hydrophilic form and a 64-kDa amphiphilic form which can be both membrane-bound and soluble. J. Biol. Chem. 1991; 266: 21257–21264.
4. Okada Y, Hosoya Y, Taniguchi H. Uptake of 3H-GABA(gamma-aminobutyric acid) and 3H-leucine in the pancreatic islets and substantia nigra of the rat. Experientia 1980; 36: 131–133.
5. Reetz A, Solimena M, Matteoli M, Folli F, Takei K, De Camilli P GABA and pancreatic β-cells: colocalization of glutamic acid decarboxylase (GAD) and GABA with synaptic-like microvesicles suggests their role in GABA storage and secretion. EMBO J. 1991; 10: 1275–1284.
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