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Increased dopamine synthesis in aging substantia nigra neurons

Published:1991-09 Issue:5 Volume:12 Page:557-565
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Greenwood Carol E.,Tatton William G.,Seniuk Nadine A.,Biddle Fred G.

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference35 articles.

1. Neurotransmitters in human brain analyzed post mortem. Changes in normal aging, senile dementia and chronic alcoholism;Carlsson,1980

2. Dopaminergic neurotoxicity of MPTP in the mouse: A murine model of Parkinsonism;Duvoisin,1986

3. Catecholamine metabolism in the brains of aging male mice;Finch;Brain Res.,1973

4. Substantia nigra lesion protects against ischemic damage in the striatum;Globus;Neurosci. Lett.,1987

5. Population death causes surviving neurons to proportionally increase catecholamine synthesis;Greenwood;Soc. Neurosci. Abstr.,1989

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2. Enhanced tyrosine hydroxylase activity induces oxidative stress, causes accumulation of autotoxic catecholamine metabolites, and augments amphetamine effects in vivo;Journal of Neurochemistry;2021-06-12

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4. A sensitive, high-throughput LC-MS/MS method for measuring catecholamines in low volume serum;Analytica Chimica Acta;2018-12

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