Theoretical investigation of the mechanism of heart failure using a canine ventricular cell model: Especially the role of up-regulated CaMKII and SR Ca2+ leak-Reference-Cited by-同舟云学术

Theoretical investigation of the mechanism of heart failure using a canine ventricular cell model: Especially the role of up-regulated CaMKII and SR Ca2+ leak

Published:2013-03 Issue: Volume:56 Page:34-43
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Zang Yunliang,Dai Ling,Zhan Heqing,Dou Jianhong,Xia Ling,Zhang Henggui

Funder

973 National Basic Research & Development Program

National Natural Science Foundation of China

Guangzhou Zhujiang Scientific and Technological New Star Capitals

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference42 articles.

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2. Transmural action potential and ionic current remodeling in ventricles of failing canine hearts;Li;Am J Physiol Heart Circ Physiol,2002

3. Heart failure enhances susceptibility to arrhythmogenic cardiac alternans;Wilson;Heart Rhythm,2009

4. Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels;Wagner;J Clin Invest,2006

5. Modulation of late sodium current by Ca2+, calmodulin, and CaMKII in normal and failing dog cardiomyocytes: similarities and differences;Maltsev;Am J Physiol Heart Circ Physiol,2008

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