Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte death-Reference-Cited by-同舟云学术

Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte death

Published:2014-10 Issue: Volume:75 Page:141-151
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Yoshida Tadashi,Friehs Ingeborg,Mummidi Srinivas,del Nido Pedro J.,Addulnour-Nakhoul Solange,Delafontaine Patrice,Valente Anthony J.,Chandrasekar Bysani

Funder

U.S. Department of Veterans Affairs

Office of Research and Development

Biomedical Laboratory Research and Development

NIH/National Heart, Lung, and Blood Institute

NIH/NIGMS

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference41 articles.

1. Interleukin-18 is a pro-hypertrophic cytokine that acts through a phosphatidylinositol 3-kinase-phosphoinositide-dependent kinase-1-Akt-GATA4 signaling pathway in cardiomyocytes;Chandrasekar;J Biol Chem,2005