TRAF3IP2 mediates interleukin-18-induced cardiac fibroblast migration and differentiation-Reference-Cited by-同舟云学术

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TRAF3IP2 mediates interleukin-18-induced cardiac fibroblast migration and differentiation

Published:2013-11 Issue:11 Volume:25 Page:2176-2184
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Valente Anthony J.,Sakamuri Siva S.V.P.,Siddesha Jalahalli M.,Yoshida Tadashi,Gardner Jason D.,Prabhu Ramesh,Siebenlist Ulrich,Chandrasekar Bysani

Funder

Department of Veterans Affairs

VA Office of Research and Development Biomedical Laboratory

NIH/NHLBI

Publisher

Elsevier BV

Subject

Cell Biology

Reference30 articles.

1. Fibroblasts in post-infarction inflammation and cardiac repair

2. Cardiac Fibroblast

3. Cardiac interstitium in health and disease: The fibrillar collagen network

4. THE CARDIAC FIBROBLAST: Therapeutic Target in Myocardial Remodeling and Failure

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1. Targeting TRAF3IP2 alleviates high glucose‐induced cardiomyocyte inflammation and apoptosis;Drug Development Research;2021-07-14

2. The SGLT2 inhibitor Empagliflozin attenuates interleukin-17A-induced human aortic smooth muscle cell proliferation and migration by targeting TRAF3IP2/ROS/NLRP3/Caspase-1-dependent IL-1β and IL-18 secretion;Cellular Signalling;2021-01

3. Empagliflozin reduces high glucose-induced oxidative stress and miR-21-dependent TRAF3IP2 induction and RECK suppression, and inhibits human renal proximal tubular epithelial cell migration and epithelial-to-mesenchymal transition;Cellular Signalling;2020-04

4. Anti-fibrotic effects of curcumin and some of its analogues in the heart;Heart Failure Reviews;2019-09-11

5. Metformin and its anti-inflammatory and anti-oxidative effects; new concepts;Journal of Renal Injury Prevention;2018-09-06

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