β-adrenergic regulation of late Na+ current during cardiac action potential is mediated by both PKA and CaMKII-Reference-Cited by-同舟云学术

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β-adrenergic regulation of late Na+ current during cardiac action potential is mediated by both PKA and CaMKII

Published:2018-10 Issue: Volume:123 Page:168-179
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Hegyi Bence,Bányász Tamás,Izu Leighton T.,Belardinelli Luiz,Bers Donald M.,Chen-Izu Ye

Funder

National Institute of Health

American Heart Association

Hungarian Scientific Research Fund

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference76 articles.

1. Induction of persistent sodium current by exogenous and endogenous nitric oxide;Ahern;J. Biol. Chem.,2000

2. Na+ channel regulation by Ca2+/calmodulin and Ca2+/calmodulin-dependent protein kinase II in guinea-pig ventricular myocytes;Aiba;Cardiovasc. Res.,2010

3. Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sites;Ashpole;J. Biol. Chem.,2012

4. Steady-state Ttx-sensitive (window) sodium current in cardiac purkinje-fibers;Attwell;Pflugers Arch.,1979

5. Eleclazine, an inhibitor of the cardiac late sodium current, is superior to flecainide in suppressing catecholamine-induced ventricular tachycardia and T-wave alternans in an intact porcine model;Bacic;Heart Rhythm.,2017

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