Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1.5 Gating by Multiple Phosphorylation Sites-Reference-Cited by-同舟云学术

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Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1.5 Gating by Multiple Phosphorylation Sites

Published:2012-06 Issue:24 Volume:287 Page:19856-19869
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Ashpole Nicole M.,Herren Anthony W.,Ginsburg Kenneth S.,Brogan Joseph D.,Johnson Derrick E.,Cummins Theodore R.,Bers Donald M.,Hudmon Andy

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference50 articles.

1. Sodium channel mutations and arrhythmias;Ruan;Nat. Rev. Cardiol.,2009

2. Two distinct congenital arrhythmias evoked by a multidysfunctional Na+ channel;Veldkamp;Circ. Res.,2000

3. Roles and regulation of the cardiac sodium channel NaV1.5. Recent insights from experimental studies;Abriel;Cardiovasc. Res.,2007

4. Identification and expression of δ-isoforms of the multifunctional Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human myocardium;Hoch;Circ. Res.,1999

5. Activity of cAMP-dependent protein kinase and Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human hearts;Kirchhefer;Cardiovasc. Res.,1999

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