Regulation of NLRP3 inflammasome by CD38 through cADPR-mediated Ca2+ release in vascular smooth muscle cells in diabetic mice-Reference-Cited by-同舟云学术

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Regulation of NLRP3 inflammasome by CD38 through cADPR-mediated Ca2+ release in vascular smooth muscle cells in diabetic mice

Published:2020-08 Issue: Volume:255 Page:117758
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Li Jia-Peng,Wei Wei,Li Xiao-Xue,Xu Ming

Funder

National Natural Science Foundation of China

“Double First-Class” University

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference58 articles.

1. Role of NLRP3 Inflammasome in the Pathogenesis of Cardiovascular Diseases;Liu,2017

2. Endothelial cells’ biophysical, biochemical, and chromosomal aberrancies in high-glucose condition within the diabetic range;Rezabakhsh;Cell Biochem. Funct.,2017

3. Quercetin alleviates high glucose-induced damage on human umbilical vein endothelial cells by promoting autophagy;Rezabakhsh;Phytomedicine,2019

4. Kruppel-like factor 15 is critical for vascular inflammation;Lu;J. Clin. Invest.,2013

5. Inflammasomes;de Zoete;Cold Spring Harb. Perspect. Biol.,2014

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1. Regulated vascular smooth muscle cell death in vascular diseases;Cell Proliferation;2024-06-14

2. CD38 deficient mice are not protected from atherosclerosis;Biochemical and Biophysical Research Communications;2024-04

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4. The Research Progress of Mitochondrial Transplantation in the Treatment of Mitochondrial Defective Diseases;International Journal of Molecular Sciences;2024-01-18

5. The NADase CD38 is a central regulator in gouty inflammation and a novel druggable therapeutic target;2023-10-23

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