Somatic mitochondrial DNA mutations do not increase neuronal vulnerability to MPTP in young POLG mutator mice-Reference-Cited by-同舟云学术

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Somatic mitochondrial DNA mutations do not increase neuronal vulnerability to MPTP in young POLG mutator mice

Published:2014-11 Issue: Volume:46 Page:62-67
ISSN:0892-0362
Container-title:Neurotoxicology and Teratology
language:en
Short-container-title:Neurotoxicology and Teratology

Author:

Dai Ying,Clark Joanne,Zheng Kangni,Kujoth Gregory C.,Prolla Tomas A.,Simon David K.

Funder

National Institute on Aging

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Developmental Neuroscience,Toxicology

Reference22 articles.

1. Expanding insights of mitochondrial dysfunction in Parkinson's disease;Abou-Sleiman;Nat Rev Neurosci,2006

2. High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease;Bender;Nat Genet,2006

3. Pgc-1 alpha overexpression downregulates Pitx3 and increases susceptibility to MPTP toxicity associated with decreased Bdnf;Clark;PLoS One,2012

4. Age-dependent cardiomyopathy in mitochondrial mutator mice is attenuated by overexpression of catalase targeted to mitochondria;Dai;Aging Cell,2010

5. Behavioral and metabolic characterization of heterozygous and homozygous POLG mutator mice;Dai;Mitochondrion,2013

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