ATF6α Optimizes Long-Term Endoplasmic Reticulum Function to Protect Cells from Chronic Stress-Reference-Cited by-同舟云学术

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ATF6α Optimizes Long-Term Endoplasmic Reticulum Function to Protect Cells from Chronic Stress

Published:2007-09 Issue:3 Volume:13 Page:351-364
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Wu Jun,Rutkowski D. Thomas,Dubois Meghan,Swathirajan Jayanth,Saunders Thomas,Wang Junying,Song Benbo,Yau Grace D.-Y.,Kaufman Randal J.

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference52 articles.

1. Endoplasmic reticulum stress induction of the Grp78/BiP promoter: Activating mechanisms mediated by YY1 and its interactive chromatin modifiers;Baumeister;Mol. Cell. Biol.,2005

2. IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA;Calfon;Nature,2002

3. The differentiation and stress response factor XBP-1 drives multiple myeloma pathogenesis;Carrasco;Cancer Cell,2007

4. Complexes containing activating transcription factor (ATF)/cAMP-responsive-element-binding protein (CREB) interact with the CCAAT/enhancer-binding protein (C/EBP)-ATF composite site to regulate Gadd153 expression during the stress response;Fawcett;Biochem. J.,1999

5. Constitutive nucleosome depletion and ordered factor assembly at the GRP78 promoter revealed by single molecule footprinting;Gal-Yam;PLoS Gen.,2006

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