Pharmacological blockade of the EP3 prostaglandin E2 receptor in the setting of type 2 diabetes enhances β-cell proliferation and identity and relieves oxidative damage-Reference-Cited by-同舟云学术

Pharmacological blockade of the EP3 prostaglandin E2 receptor in the setting of type 2 diabetes enhances β-cell proliferation and identity and relieves oxidative damage

Published:2021-12 Issue: Volume:54 Page:101347
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Bosma Karin J.^ORCID,Andrei Spencer R.,Katz Liora S.^ORCID,Smith Ashley A.^ORCID,Dunn Jennifer C.^ORCID,Ricciardi Valerie F.^ORCID,Ramirez Marisol A.^ORCID,Baumel-Alterzon Sharon^ORCID,Pace William A.^ORCID,Carroll Darian T.^ORCID,Overway Emily M.^ORCID,Wolf Elysa M.^ORCID,Kimple Michelle E.^ORCID,Sheng Quanhu,Scott Donald K.,Breyer Richard M.^ORCID,Gannon Maureen^ORCID

Funder

Israel National Road Safety Authority

National Institutes of Health

National Institute of Diabetes and Digestive and Kidney Diseases

U.S. Department of Veterans Affairs

Vanderbilt University

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference71 articles.

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3. Islet G protein-coupled receptors as potential targets for treatment of type 2 diabetes;Ahren;Nature Reviews Drug Discovery,2009

4. Gastrointestinal hormones and the regulation of beta-cell mass;Lavine;Annals of the New York Academy of Sciences,2010

5. Determining predictors of response to exenatide in type 2 diabetes;Anderson;Journal of the American Pharmacists Association (2003),2012

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