Systemic administration of scAAV9-IGF1 extends survival in SOD1G93A ALS mice via inhibiting p38 MAPK and the JNK-mediated apoptosis pathway-Reference-Cited by-同舟云学术

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Systemic administration of scAAV9-IGF1 extends survival in SOD1G93A ALS mice via inhibiting p38 MAPK and the JNK-mediated apoptosis pathway

Published:2018-05 Issue: Volume:139 Page:203-210
ISSN:0361-9230
Container-title:Brain Research Bulletin
language:en
Short-container-title:Brain Research Bulletin

Author:

Wang Wan,Wen Di,Duan Weisong,Yin Juntao,Cui Can,Wang Ying,Li Zhongyao,Liu Yakun,Li Chunyan

Funder

Scientific Research Fund of The Second Hospital of Hebei Medical University

Publisher

Elsevier BV

Subject

General Neuroscience

Reference40 articles.

1. A unifying hypothesis for the cause of amyotrophic lateral sclerosis, parkinsonism, and Alzheimer disease;Appel;Ann. Neurol.,1981

2. Induction of functional neovascularization by combined VEGF and angiopoietin-1 gene transfer using AAV vectors;Arsic;Mol. Ther.,2003

3. Inhibition of p38 mitogen-activated protein kinase provides neuroprotection in cerebral focal ischemia;Barone;Med. Res. Rev.,2001

4. Identification of a novel Bcl-xL phosphorylation site regulating the sensitivity of taxol- or 2-methoxyestradiol-induced apoptosis;Basu;FEBS Lett.,2003

5. Genetic correction of SOD1 mutant iPSCs reveals ERK and JNK activated AP1 as a driver of neurodegeneration in amyotrophic lateral sclerosis;Bhinge;Stem Cell Rep.,2017

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