Fragile X Protein is required for inhibition of insulin signaling and regulates glial-dependent neuroblast reactivation in the developing brain-Reference-Cited by-同舟云学术

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Fragile X Protein is required for inhibition of insulin signaling and regulates glial-dependent neuroblast reactivation in the developing brain

Published:2012-06 Issue: Volume:1462 Page:151-161
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Callan Matthew A.,Clements Nicole,Ahrendt Nicholas,Zarnescu Daniela C.

Funder

Arizona Biomedical Research Commission

Department of Defense Idea Award

Jim Himelic Foundation

Howard Hughes Medical Institute

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference44 articles.

1. Neural stem cell biology in vertebrates and invertebrates: more alike than different?;Brand;Neuron,2011

2. Targeted gene expression as a means of altering cell fates and generating dominant phenotypes;Brand;Development,1993

3. Heads-up: new roles for the fragile X mental retardation protein in neural stem and progenitor cells;Callan;Genesis,2011

4. Fragile X protein controls neural stem cell proliferation in the Drosophila brain;Callan;Hum. Mol. Genet.,2010

5. Altered differentiation of neural stem cells in fragile X syndrome;Castren;Proc. Natl. Acad. Sci. U. S. A.,2005

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1. Role of fragile X messenger ribonucleoprotein 1 in the pathophysiology of brain disorders: a glia perspective;Neuroscience & Biobehavioral Reviews;2024-07

2. PGC-1α integrates insulin signaling with mitochondrial physiology and behavior in a Drosophila model of Fragile X Syndrome;npj Metabolic Health and Disease;2024-02-21

3. Dysregulation of BMP, Wnt, and Insulin Signaling in Fragile X Syndrome;Frontiers in Cell and Developmental Biology;2022-07-06

4. Loss of cytoplasmic FMR1‐interacting protein 2 (CYFIP2) induces browning in 3T3‐L1 adipocytes via repression of GABA‐BR and activation of mTORC1;Journal of Cellular Biochemistry;2022-03

5. Non-autonomous regulation of neurogenesis by extrinsic cues: a Drosophila perspective;Oxford Open Neuroscience;2022

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