Inhibition of mitochondrial permeability transition pore opening contributes to the neuroprotective effects of ischemic postconditioning in rats-Reference-Cited by-同舟云学术

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Inhibition of mitochondrial permeability transition pore opening contributes to the neuroprotective effects of ischemic postconditioning in rats

Published:2012-02 Issue: Volume:1436 Page:101-110
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Sun Jing,Luan Qi,Dong Hailong,Song Wenying,Xie Keliang,Hou Lichao,Xiong Lize

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference34 articles.

1. The role of mitochondrial permeability transition pore in transmembrane Ca2+-exchange in mitochondria;Akopova;Ukr. Biokhim. Zh.,2008

2. Core and penumbral nitric oxide synthase activity during cerebral ischemia and reperfusion;Ashwal;Stroke,1998

3. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death;Baines;Nature,2005

4. Phosphate is essential for inhibition of the mitochondrial permeability transition pore by cyclosporin A and by cyclophilin D ablation;Basso;J. Biol. Chem.,2008

5. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding;Bradford;Anal. Biochem.,1976

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