Silencing BMAL1 promotes M1/M2 polarization through the LDHA/lactate axis to promote GBM sensitivity to bevacizumab-Reference-Cited by-同舟云学术

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Silencing BMAL1 promotes M1/M2 polarization through the LDHA/lactate axis to promote GBM sensitivity to bevacizumab

Published:2024-06 Issue: Volume:134 Page:112187
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Wang Fan,Liao Wenjun,Li Caiyan,Zhu Ling^ORCID

Publisher

Elsevier BV

Reference44 articles.

1. Clonal ZEB1-Driven Mesenchymal Transition Promotes Targetable Oncologic Antiangiogenic Therapy Resistance;Chandra;Cancer Res.,2020

2. Management of paediatric glioblastoma;Pervez;J Pak Med Assoc.,2021

3. Aryl hydrocarbon receptor nuclear translocator-like (ARNTL/BMAL1) is associated with bevacizumab resistance in colorectal cancer via regulation of vascular endothelial growth factor A;Burgermeister;EBioMedicine.,2019

4. GBP3 promotes glioblastoma resistance to temozolomide by enhancing DNA damage repair;Xu;Oncogene.,2022

5. Dendritic Cell Vaccination of Glioblastoma: Road to Success or Dead End;Datsi;Front Immunol.,2021

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