Optimization of dietary folate or low-dose folic acid supplements lower homocysteine but do not enhance endothelial function in healthy adults, irrespective of the methylenetetrahydrofolate reductase (C677T) genotype-Reference-Cited by-同舟云学术

Optimization of dietary folate or low-dose folic acid supplements lower homocysteine but do not enhance endothelial function in healthy adults, irrespective of the methylenetetrahydrofolate reductase (C677T) genotype

Published:2001-12 Issue:7 Volume:38 Page:1799-1805
ISSN:0735-1097
Container-title:Journal of the American College of Cardiology
language:en
Short-container-title:Journal of the American College of Cardiology

Author:

Pullin Catherine H,Ashfield-Watt Pauline A.L,Burr Michael L,Clark Zoë E,Lewis Malcolm J,Moat Stuart J,Newcombe Robert G,Powers Hilary J,Whiting Jenny M,McDowell Ian F.W

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference35 articles.

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2. Plasma homocysteine and coronary heart disease;Danesh;J Cardiovasc Risk,1998

3. Vitamin status and intake as primary determinants of homocysteinemia in an elderly population;Selhub;JAMA,1993

4. Lowering blood homocysteine with folic acid–based supplements;BMJ,1998

5. Relationship between folate status, a common mutation in methylenetetrahydrofolate reductase, and plasma homocysteine concentrations;Jacques;Circulation,1996

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