HSP27 expression in primary colorectal cancers is dependent on mutation of KRAS and PI3K/AKT activation status and is independent of TP53-Reference-Cited by-同舟云学术

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HSP27 expression in primary colorectal cancers is dependent on mutation of KRAS and PI3K/AKT activation status and is independent of TP53

Published:2013-02 Issue:1 Volume:94 Page:103-108
ISSN:0014-4800
Container-title:Experimental and Molecular Pathology
language:en
Short-container-title:Experimental and Molecular Pathology

Author:

Ghosh Anil,Lai Cecilia,McDonald Sarah,Suraweera Nirosha,Sengupta Neel,Propper David,Dorudi Sina,Silver Andrew

Funder

Constance Travis Charitable Trust

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Pathology and Forensic Medicine

Reference42 articles.

1. Clinicopathological analysis of colorectal cancers with PIK3CA mutations in Middle Eastern population;Abubaker;Oncogene,2008

2. Kirsten ras mutations in patients with colorectal cancer: the ‘RASCAL II’ study;Andreyev;British Journal of Cancer,2001

3. Clinicopathologic significance of BAG1 and TIMP3 expression in colon carcinoma;Bai;World Journal of Gastroenterology,2007

4. Mutations in the RAS-MAPK, PI(3)K (phosphatidylinositol-3-OH kinase) signaling network correlate with poor survival in a population-based series of colon cancers;Barault;International Journal of Cancer,2008

5. Molecular chaperones and protein quality control;Bukau;Cell,2006

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3. The role of heat shock proteins in metastatic colorectal cancer: A review;Journal of Cellular Biochemistry;2022-09-05

4. The Role of Heat Shock Protein 27 in Carcinogenesis and Treatment of Colorectal Cancer;Current Pharmaceutical Design;2022-09

5. p16 is superior to Stathmin-1 and HSP27 in identifying cervical dysplasia;Diagnostic Pathology;2021-09-20

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