Multiple deficiencies in antioxidant enzymes in mice result in a compound increase in sensitivity to oxidative stress-Reference-Cited by-同舟云学术

Multiple deficiencies in antioxidant enzymes in mice result in a compound increase in sensitivity to oxidative stress

Published:2004-06 Issue:12 Volume:36 Page:1625-1634
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Van Remmen Holly,Qi Wenbo,Sabia Marian,Freeman Gregory,Estlack Larry,Yang Hong,Mao Guo Zhong,Huang Ting-Ting,Strong Randy,Lee Shuko,Epstein Charles J.,Richardson Arlan

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference41 articles.

1. Mice lacking extracellular superoxide dismutase are more sensitive to hyperoxia;Carlsson;Proc. Natl. Acad. Sci. USA,1995

2. Edema formation exacerbates neurological and histological outcomes after focal cerebral ischemia in CuZn-superoxide dismutase gene knockout mutant mice;Kondo;Acta Neurochir. Suppl. (Wien),1997

3. Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase;Li;Nat. Genet.,1995

4. Mice with a homozygous null mutation for the most abundant glutathione peroxidase, Gpx1, show increased susceptibility to the oxidative stress-inducing agents paraquat and hydrogen peroxide;De Haan;J. Biol. Chem.,1998

5. Mice deficient in cellular glutathione peroxidase develop normally and show no increased sensitivity to hyperoxia;Ho;J. Biol. Chem.,1997

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