Reduced plasticity and mild cognitive impairment-like deficits after entorhinal lesions in hAPP/APOE4 mice-Reference-Cited by-同舟云学术

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Reduced plasticity and mild cognitive impairment-like deficits after entorhinal lesions in hAPP/APOE4 mice

Published:2013-11 Issue:11 Volume:34 Page:2683-2693
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Bott Jean-Bastien,Cosquer Brigitte,Héraud Céline,Zerbinatti Celina,Kelche Christian,Cassel Jean-Christophe,Mathis Chantal

Funder

Alsace Alzheimer Haut-Rhin (68)

University of Strasbourg

Centre National de la Recherche Scientifique

Ministère de l'Enseignement Supérieur et de la Recherche

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference67 articles.

1. Plastic neuronal remodeling is impaired in patients with Alzheimer's disease carrying apolipoprotein ε4 allele;Arendt;J. Neurosci.,1997

2. APOE, ACT and CHRNA7 genes in the conversion from amnestic mild cognitive impairment to Alzheimer's disease;Barabash;Neurobiol. Aging,2009

3. A deficit in astroglial organization causes the impaired reactive sprouting in human apolipoprotein E4 targeted replacement mice;Blain;Neurobiol. Dis.,2006

4. Middle-aged human apoE4 targeted-replacement mice show retention deficits on a wide range of spatial memory tasks;Bour;Behav. Brain Res.,2008

5. No evidence for cholinergic problems in apolipoprotein E knockout and apolipoprotein E4 transgenic mice;Bronfman;Neuroscience,2000

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