Hepatocytic AP-1 and STAT3 contribute to chemotaxis in alphanaphthylisothiocyanate-induced cholestatic liver injury-Reference-Cited by-同舟云学术

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Hepatocytic AP-1 and STAT3 contribute to chemotaxis in alphanaphthylisothiocyanate-induced cholestatic liver injury

Published:2023-01 Issue: Volume:373 Page:184-193
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Luo Yishuang,Kang Jinyu,Luo Jia,Yan Zheng,Li Shengtao,Lu Zhuoheng,Song Yufei,Zhang Xie,Yang Julin,Liu Aiming

Funder

Zhejiang Province Basic Public Welfare Research Program

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference41 articles.

1. Kupffer cell blockade improves the endotoxin-induced microcirculatory inflammatory response in obstructive jaundice;Abrahám;Shock,2008

2. Bile acids induce inflammatory genes in hepatocytes: a novel mechanism of inflammation during obstructive cholestasis;Allen;Am. J. Pathol.,2011

3. Neutrophil function: from mechanisms to disease;Amulic;Annu Rev. Immunol.,2012

4. The role of inflammation in the mechanisms of bile acid-induced liver damage;Cai;Dig. Dis.,2017

5. Bile acids initiate cholestatic liver injury by triggering a hepatocyte-specific inflammatory response;Cai;JCI Insight,2017

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