2,3′4,4′,5-Pentachlorobiphenyl induces hepatocellular carcinoma cell proliferation through pyruvate kinase M2-dependent glycolysis-Reference-Cited by-同舟云学术

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2,3′4,4′,5-Pentachlorobiphenyl induces hepatocellular carcinoma cell proliferation through pyruvate kinase M2-dependent glycolysis

Published:2019-10 Issue: Volume:313 Page:108-119
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Liang Wenli,Zhang Yuting,Song Li,Li Zhuoyu

Funder

National Natural Sciences Foundation of China

Natural Sciences Foundation of Shanxi

University Science and technology innovation project of Shanxi

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference61 articles.

1. Hexabromocyclododecane and polychlorinated biphenyls increase resistance of hepatocellular carcinoma cells to cisplatin through the phosphatidylinositol 3-kinase/protein kinase B pathway;An;Toxicol. Lett.,2014

2. The chlorinated AHR ligand 3,3’,4,4′,5-pentachlorobiphenyl (PCB126) promotes reactive oxygen species (ROS) production during embryonic development in the killifish (Fundulus heteroclitus);Arzuaga;Aquat. Toxicol.,2006

3. Hormesis: U-shaped dose responses and their centrality in toxicology;Calabrese;Trends Pharmacol. Sci.,2001

4. Epidemiological characteristics and risk factors of hepatocellular carcinoma;Chen;J. Gastroenterol. Hepatol.,1997

5. Mechanisms of pyruvate kinase M2 isoform inhibits cell motility in hepatocellular carcinoma cells;Chen;World J. Gastroenterol.,2015

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