P2x7 receptors control demyelination and inflammation in the cuprizone model-Reference-Cited by-同舟云学术

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P2x7 receptors control demyelination and inflammation in the cuprizone model

Published:2020-04 Issue: Volume:4 Page:100062
ISSN:2666-3546
Container-title:Brain, Behavior, & Immunity - Health
language:en
Short-container-title:Brain, Behavior, & Immunity - Health

Author:

Bernal-Chico Ana,Manterola Andrea,Cipriani Raffaela,Katona István,Matute Carlos,Mato Susana

Funder

Spanish Ministry of Economy and Competitiveness

CIBERNED

ISCIII

Basque Government

ARSEP Foundation

Hungarian Academy of Sciences

Momentum Program

Janssen Research & Development

Publisher

Elsevier BV

Subject

General Medicine

Reference71 articles.

1. NAD+ released during inflammation participates in T cell homeostasis by inducing ART2-mediated death of naive T cells in vivo;Adriouch;J. Immunol.,2007

2. Progesterone therapy induces an M1 to M2 switch in microglia phenotype and suppresses NLRP3 inflammasome in a cuprizone-induced demyelination mouse model;Aryanpour;Int. Immunopharm.,2017

3. High sensitivity of CD4+CD25+ regulatory T cells to extracellular metabolites nicotinamide adenine dinucleotide and ATP: a role for P2X7 receptors;Aswad;J. Immunol.,2005

4. Inflammasome activation in multiple sclerosis and experimental autoimmune encephalomyelitis (EAE);Barclay;Brain Pathol.,2017

5. Increased proliferation rate of lymphoid cells transfected with the P2X(7) ATP receptor;Baricordi;J. Biol. Chem.,1999

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3. Partners in health and disease: pineal gland and purinergic signalling;Purinergic Signalling;2024-07-20

4. N-butyl-β-carboline-3-carboxylate (β-CCB) systemic administration promotes remyelination in the cuprizone demyelinating model in mice;Scientific Reports;2024-06-18

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