Activation of c-Jun N-terminal kinase (JNK) by widely used specific p38 MAPK inhibitors SB202190 and SB203580: A MLK-3-MKK7-dependent mechanism
Author:
Publisher
Elsevier BV
Subject
Cell Biology
Reference24 articles.
1. p38 MAP kinases: key signalling molecules as therapeutic targets for inflammatory diseases
2. From JNK to Pay Dirt: Jun Kinases, their Biochemistry, Physiology and Clinical Importance
3. Signal Transduction by the JNK Group of MAP Kinases
4. MAPK signalling pathways as molecular targets for anti-inflammatory therapy—from molecular mechanisms to therapeutic benefits
5. Inflammation-activated Protein Kinases as Targets for Drug Development
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