TGF-β/NF1/Smad4-mediated suppression of ANT2 contributes to oxidative stress in cellular senescence-Reference-Cited by-同舟云学术

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TGF-β/NF1/Smad4-mediated suppression of ANT2 contributes to oxidative stress in cellular senescence

Published:2014-12 Issue:12 Volume:26 Page:2903-2911
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Kretova Miroslava,Sabova Ludmila,Hodny Zdenek,Bartek Jiri,Kollarovic Gabriel,Nelson Buck D.,Hubackova Sona,Luciakova Katarina

Funder

Slovak Grant Agency VEGA

Grant Agency of the Czech Republic

Institutional Grant

Swedish Science Council

Publisher

Elsevier BV

Subject

Cell Biology

Reference73 articles.

1. NEW EMBO MEMBERS REVIEW: Transcriptional control by the TGF-beta/Smad signaling system

2. SPECIFICITY AND VERSATILITY IN TGF-β SIGNALING THROUGH SMADS

3. Mechanism of TGF-β signaling to growth arrest, apoptosis, and epithelial–mesenchymal transition

4. Tissue-specific transcription pattern of the adenine nucleotide translocase isoforms in humans

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1. Emerging Role of Fibroblasts in Vitiligo: A Formerly Underestimated Rising Star;Journal of Investigative Dermatology;2024-08

2. Loss of ANT1 Increases Fibrosis and Epithelial Cell Senescence in Idiopathic Pulmonary Fibrosis;American Journal of Respiratory Cell and Molecular Biology;2023-11

3. DNA‐PKcs and ATM modulate mitochondrial ADP‐ATP exchange as an oxidative stress checkpoint mechanism;The EMBO Journal;2023-02-02

4. Loss of ANT1 Increases Fibrosis and Epithelial Cell Senescence in Idiopathic Pulmonary Fibrosis;2022-09-09

5. Temporal changes in mitochondrial function and reactive oxygen species generation during the development of replicative senescence in human fibroblasts;Experimental Gerontology;2022-08

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