Japanese Encephalitis Virus infection increases USP42 to stabilize TRIM21 and OAS1 for neuroinflammatory and anti-viral response in human microglia-Reference-Cited by-同舟云学术

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Japanese Encephalitis Virus infection increases USP42 to stabilize TRIM21 and OAS1 for neuroinflammatory and anti-viral response in human microglia

Published:2022-08 Issue: Volume:573 Page:131-140
ISSN:0042-6822
Container-title:Virology
language:en
Short-container-title:Virology

Author:

Mishra Ritu,Kumawat Kanhaiya Lal,Basu Anirban,Banerjea Akhil C.

Funder

National Institute of Immunology

Department of Science and Technology, Ministry of Science and Technology, India

Amity University

Science and Engineering Research Board

Publisher

Elsevier BV

Subject

Virology

Reference61 articles.

1. Enhancement of Th1/Th17 inflammation by TRIM21 in Behçet’s disease;Ahn;Sci. Rep.,2017

2. Pathogenicity and virulence of Japanese encephalitis virus: neuroinflammation and neuronal cell damage;Ashraf;Virulence,2021

3. Involvement of RIG-ipathway in neurotropic virus-induced acute flaccid paralysis and subsequent spinal motor neuron death;Bhaskar 1;mBio,2021

4. The ubiquitin-conjugating system: multiple roles in viral replication and infection;Calistri;Cells,2014

5. Glial activation involvement in neuronal death by Japanese encephalitis virus infection;Chen;J. Gen. Virol.,2010

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Viral infection and spread are inhibited by the polyubiquitination and downregulation of TRPV2 channel by the interferon-stimulated gene TRIM21;Cell Reports;2024-04

2. Friend or foe? Reciprocal regulation between E3 ubiquitin ligases and deubiquitinases;Biochemical Society Transactions;2024-02-28

3. Blocking of doublecortin-like kinase 1-regulated SARS-CoV-2 replication cycle restores cell signaling network;Journal of Virology;2023-11-30

4. OAS1 suppresses African swine fever virus replication by recruiting TRIM21 to degrade viral major capsid protein;Journal of Virology;2023-10-31

5. TRIM21 Promotes Rabies Virus Production by Degrading IRF7 through Ubiquitination;International Journal of Molecular Sciences;2023-06-30

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