Virus infection rapidly activates the P58IPK pathway, delaying peak kinase activation to enhance viral replication-Reference-Cited by-同舟云学术

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Virus infection rapidly activates the P58IPK pathway, delaying peak kinase activation to enhance viral replication

Published:2011-08 Issue:1 Volume:417 Page:27-36
ISSN:0042-6822
Container-title:Virology
language:en
Short-container-title:Virology

Author:

Goodman Alan G.,Tanner Bertrand C.W.,Chang Stewart T.,Esteban Mariano,Katze Michael G.

Funder

Ministry of Science and Education of Spain Program JAE-DOC

National Science Foundation Postdoctoral Fellowship in Biology

Marcelino Botín Foundation

National Institute of Allergy and Infectious Diseases

National Institutes of Health

Department of Health and Human Services

Public Health Service

Publisher

Elsevier BV

Subject

Virology

Reference43 articles.

1. Host-range restriction of vaccinia virus E3L-specific deletion mutants;Beattie;Virus Gene,1996

2. Influenza virus NS1 protein counteracts PKR-mediated inhibition of replication;Bergmann;J. Virol.,2000

3. Antiviral effect of the mammalian translation initiation factor 2[alpha] kinase GCN2 against RNA viruses;Berlanga;EMBO J.,2006

4. Sensitivity and uncertainty analysis of complex models of disease transmission: an HIV model, as an example;Blower;Int. Stat. Rev.,1994

5. Deoxyribonucleic acid-dependent ribonucleic acid polymerase activity in cells infected with influenza virus;Borland;J. Virol.,1968

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