Membrane protein carbonylation of Plasmodium falciparum infected erythrocytes under conditions of sickle cell trait and G6PD deficiency-Reference-Cited by-同舟云学术

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Membrane protein carbonylation of Plasmodium falciparum infected erythrocytes under conditions of sickle cell trait and G6PD deficiency

Published:2019-01 Issue: Volume:227 Page:5-14
ISSN:0166-6851
Container-title:Molecular and Biochemical Parasitology
language:en
Short-container-title:Molecular and Biochemical Parasitology

Author:

Contreras-Puentes Neyder^ORCID,Rodríguez-Cavallo Erika^ORCID,Méndez-Cuadro Darío^ORCID

Funder

Departamento Administrativo de Ciencia, Tecnología e Investigación — COLCIENCIAS [Administrative Department of Science, Technology and Research]

The University of Cartagena

Publisher

Elsevier BV

Subject

Molecular Biology,Parasitology

Reference51 articles.

1. G6PD deficiency: the genotype-phenotype association;Mason;Blood Rev.,2007

2. Glucose-6-phosphate dehydrogenase deficiency and malaria: cytochemical detection of heterozygous G6PD deficiency in women;Peters;J. Histochem. Cytochem.,2009

3. A cost-effectiveness analysis of a pilot neonatal screening program for sickle cell Anemia in the Republic of Angola;McGann;J. Pediatr.,2015

4. Biochemical and immunological mechanisms by which sickle cell trait protects against malaria;Gong;Malar. J.,2013

5. Changes in red blood cell membrane structure in G6PD deficiency: an atomic force microscopy study;Tang;Clin. Chim. Acta,2015

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4. Erythrocyte membrane proteins involved in the immune response to Plasmodium falciparum and Plasmodium vivax infection;Parasitology Research;2021-04-02

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