Compromising KCC2 transporter activity enhances the development of continuous seizure activity-Reference-Cited by-同舟云学术

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Compromising KCC2 transporter activity enhances the development of continuous seizure activity

Published:2016-09 Issue: Volume:108 Page:103-110
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Kelley Matthew R.^ORCID,Deeb Tarek Z.,Brandon Nicholas J.,Dunlop John,Davies Paul A.,Moss Stephen J.

Funder

Simons Foundation Grant

National Institutes of Health/National Institute of Neurological Disorders and Stroke Grants

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference48 articles.

1. The contribution of raised intraneuronal chloride to epileptic network activity;Alfonsa;J. Neurosci.,2015

2. Magnesium-free medium activates seizure-like events in the rat hippocampal slice;Anderson;Brain Res.,1986

3. Synchronous discharges in the rat limbic potentials and epileptiform system in vitro;Avoli;J. Neurosci.,1996

4. CA3-driven hippocampal-entorhinal loop controls rather than sustains in vitro limbic seizures;Barbarosie;J. Neurosci.,1997

5. Positive shifts of the GABAA receptor reversal potential due to altered chloride homeostasis is widespread after status epilepticus;Barmashenko;Epilepsia,2011

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1. The brain-specific kinase LMTK3 regulates neuronal excitability by decreasing KCC2-dependent neuronal Cl− extrusion;iScience;2024-04

2. Neuronal K+-Cl- cotransporter KCC2 as a promising drug target for epilepsy treatment;Acta Pharmacologica Sinica;2023-09-13

3. Excitatory/inhibitory balance in epilepsies and neurodevelopmental disorders: Depolarizing γ‐aminobutyric acid as a common mechanism;Epilepsia;2023-06-04

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