The brain-specific kinase LMTK3 regulates neuronal excitability by decreasing KCC2-dependent neuronal Cl− extrusion-Reference-Cited by-同舟云学术

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The brain-specific kinase LMTK3 regulates neuronal excitability by decreasing KCC2-dependent neuronal Cl− extrusion

Published:2024-04 Issue:4 Volume:27 Page:109512
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Cho Noell,Kontou Georgina,Smalley Joshua L.,Bope Christopher,Dengler Jacob,Montrose Kristopher,Deeb Tarek Z.,Brandon Nicholas J.,Yamamoto Tadashi,Davies Paul A.,Giamas Georgios,Moss Stephen J.

Funder

National Institutes of Health

AstraZeneca

NINDS

NIMH

Publisher

Elsevier BV

Reference58 articles.

1. The K+/Cl- co-transporter KCC2 renders GABA hyperpolarizing during neuronal maturation;Rivera;Nature,1999

2. The neuron-specific K-Cl cotransporter, KCC2. Antibody development and initial characterization of the protein;Williams;J. Biol. Chem.,1999

3. Cation-chloride cotransporters in neuronal development, plasticity and disease;Kaila;Nat. Rev. Neurosci.,2014

4. The Multifaceted Roles of KCC2 in Cortical Development;Virtanen;Trends Neurosci.,2021

5. Identification of KCC2 Mutations in Human Epilepsy Suggests Strategies for Therapeutic Transporter Modulation;Duy;Front. Cell. Neurosci.,2019

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