ApoE is necessary and sufficient for the binding of large triglyceride-rich lipoproteins to the LDL receptor; apoB is unnecessary.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Endocrinology,Biochemistry
Reference33 articles.
1. Apolipoprotein E degradation in human very low density lipoproteins by plasma protease(s): chemi cal and biochemical consequences;Bradley;Biochem. Biophys. Res. Commun.,1982
2. Apolipoprotein E mediates uptake of Sf 100-400 hypertriglyceridemic very low density lipoproteins by the low density lipoprotein receptor pathway in normal human fibroblasts;Gianturco;J. Biol. Chem.,1983
3. Low density lipoprotein receptor binding determinants switch from apolipoprotein E to apolipoprotein B during conversion of hypertriglyceridemic very-low-density lipoprotein to low-density lipoproteins;Bradley;J. Biol. Chem.,1984
4. Receptor-mediated uptake of hypertriglyceridemic very low density lipoproteins by normal human fibroblasts;Gianturco;J. Lipid Res.,1982
5. Control of 3-hydroxy-3-methylglutaryl CoA reductase activity in cultured human fibroblasts by very low density lipoproteins of subjects with hypertriglyceridemia;Gianturco;J. Clin. Invest.,1978
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