Apolipoprotein-E degradation in human very low density lipoproteins by plasma protease(s): Chemical and biological consequences
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference22 articles.
1. Control of 3-Hydroxy-3-Methylglutaryl-CoA Reductase Activity in Cultured Human Fibroblasts by Very Low Density Lipoproteins of Subjects with Hypertriglyceridemia
2. Abnormal suppression of 3-hydroxy-3-methylglutaryl-CoA reductase activity in cultured human fibroblasts by hypertriglyceridemic very low density lipoprotein subclasses
3. Regulation of Plasma Cholesterol by Lipoprotein Receptors
4. Receptor-mediated uptake of hypertriglyceridemic very low density lipoproteins by normal human fibroblasts
5. Plasma lipids as collateral risk factors in coronary artery disease—A study of 371 males with chest pain
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2. Apolipoprotein E: Structural Insights and Links to Alzheimer Disease Pathogenesis;Neuron;2021-01
3. In Vivo and In Vitro Effects of an Apolipoprotein E Mimetic Peptide on Amyloid-β Pathology;Journal of Alzheimer's Disease;2013-06-18
4. IL-33/ST2 signalling contributes to carrageenin-induced innate inflammation and inflammatory pain: role of cytokines, endothelin-1 and prostaglandin E2;British Journal of Pharmacology;2013-04-12
5. Apolipoprotein E mimetic is more effective than apolipoprotein A-I mimetic in reducing lesion formation in older female apo E null mice;Atherosclerosis;2012-10
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