IL-33/ST2 signalling contributes to carrageenin-induced innate inflammation and inflammatory pain: role of cytokines, endothelin-1 and prostaglandin E2-Reference-Cited by-同舟云学术

IL-33/ST2 signalling contributes to carrageenin-induced innate inflammation and inflammatory pain: role of cytokines, endothelin-1 and prostaglandin E2

Published:2013-04-12 Issue:1 Volume:169 Page:90-101
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:Br J Pharmacol

Author:

Zarpelon AC¹,Cunha TM²,Alves-Filho JC,Pinto LG²,Ferreira SH²,McInnes IB³,Xu D³,Liew FY³,Cunha FQ²,Verri WA¹

Affiliation:

1. Departamento de Patologia; Centro de Ciências Biológicas; Universidade Estadual de Londrina; Londrina; Parana; Brazil

2. Department of Pharmacology; School of Medicine of Ribeirão Preto; University of São Paulo; Ribeirão Preto; São Paulo; Brazil

3. Division of Immunology, Infection and Inflammation; Glasgow Biomedical Research Centre; University of Glasgow; Glasgow; UK

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.12110/fullpdf

Reference59 articles.

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3. Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection;Alves-Filho;Nat Med,2010

4. Amplification of cytokine production through synergistic activation of NFAT and AP-1 following stimulation of mast cells with antigen and IL-33;Andrade;Eur J Immunol,2011

5. NKT cells are required to induce high IL-33 expression in hepatocytes during ConA-induced acute hepatitis;Arshad;Eur J Immunol,2011

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