Bradykinin decreases nitric oxide release from microglia via inhibition of cyclic adenosine monophosphate signaling-Reference-Cited by-同舟云学术

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Bradykinin decreases nitric oxide release from microglia via inhibition of cyclic adenosine monophosphate signaling

Published:2013-02 Issue: Volume:40 Page:133-140
ISSN:0196-9781
Container-title:Peptides
language:en
Short-container-title:Peptides

Author:

Ben-Shmuel Sarit,Danon Abraham,Fleisher-Berkovich Sigal

Funder

Israel Science Foundation

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrinology,Physiology,Biochemistry

Reference47 articles.

1. Inflammation in neurodegenerative diseases;Amor;Immunology,2010

2. Microglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanism;Block;Prog Neurobiol,2005

3. Bradykinin protects against brain microvascular endothelial cell death induced by pathophysiological stimuli;Bovenzi;J Cell Physiol,2010

4. Mechanisms of inflammatory neurodegeneration: iNOS and NADPH oxidase;Brown;Biochem Soc Trans,2007

5. Lipoteichoic acid-induced nitric oxide synthase expression in RAW 264.7 macrophages is mediated by cyclooxygenase-2, prostaglandin E2, protein kinase A, p38 MAPK, and nuclear factor-kappaB pathways;Chang;Cell Signal,2006

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1. Uso dos antagonistas dos receptores de bradicinina na doença de Alzheimer;Revista Contemporânea;2022-05-31

2. Microglia and bradykinin cross talk in poststroke cognitive impairment in diabetes;American Journal of Physiology-Cell Physiology;2021-04-01

3. The Dual Role of Kinin/Kinin Receptors System in Alzheimer’s Disease;Frontiers in Molecular Neuroscience;2019-10-01

4. Potential Use of Nanomedicine for the Anti-inflammatory Treatment of Neurodegenerative Diseases;Current Pharmaceutical Design;2018-07-13

5. Implication of the Kallikrein-Kinin system in neurological disorders: Quest for potential biomarkers and mechanisms;Progress in Neurobiology;2018-06

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