CCL2 is a key mediator of microglia activation in neuropathic pain states-Reference-Cited by-同舟云学术

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CCL2 is a key mediator of microglia activation in neuropathic pain states

Published:2009-03 Issue:3 Volume:13 Page:263-272
ISSN:1090-3801
Container-title:European Journal of Pain
language:en
Short-container-title:

Author:

Thacker Michael A.,Clark Anna K.,Bishop Thomas,Grist John,Yip Ping K.,Moon Lawrence D.F.,Thompson Stephen W.N.,Marchand Fabien,McMahon Stephen B.

Publisher

Wiley

Subject

Anesthesiology and Pain Medicine

Reference35 articles.

1. Impaired neuropathic pain responses in mice lacking the chemokine receptor CCR2;Abbadie;PNAS,2003

2. Local self-renewal can sustain CNS microglia maintenance and function throughout adult life;Ajami;Nat Neurosci,2007

3. Chemokine/chemokine receptor nomenclature;Bacon;J Interferon Cytokine Res,2002

4. Constitutive neuronal expression of CCR2 chemokine receptor and its colocalization with neurotransmitters in normal rat brain: functional effect of MCP-1/CCL2 on calcium mobilization in primary cultured neurons;Banisadr;J Comp Neurol,2005

5. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man;Bennett;Pain,1988

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