Glutaredoxin 2a overexpression in macrophages promotes mitochondrial dysfunction but has little or no effect on atherogenesis in LDL-receptor null mice-Reference-Cited by-同舟云学术

Glutaredoxin 2a overexpression in macrophages promotes mitochondrial dysfunction but has little or no effect on atherogenesis in LDL-receptor null mice

Published:2015-07 Issue:1 Volume:241 Page:69-78
ISSN:0021-9150
Container-title:Atherosclerosis
language:en
Short-container-title:Atherosclerosis

Author:

Zamora D.A.,Downs K.P.,Ullevig S.L.,Tavakoli S.,Kim H.S.,Qiao M.,Greaves D.R.,Asmis R.^ORCID

Funder

American Heart Association

National Institutes of Health

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference54 articles.

1. Monocyte and macrophage heterogeneity;Gordon;Nat. Rev. Immunol.,2005

2. Protective and pathogenic functions of macrophage subsets;Murray;Nat. Rev. Immunol.,2011

3. Tissue-resident macrophages;Davies;Nat. Immunol.,2013

4. Bioenergetic profiles diverge during macrophage polarization: Implications for the interpretation of 18F-FDG PET imaging of atherosclerosis;Tavakoli;J. Nucl. Med.,2013

5. A membrane protease regulates energy production in macrophages by activating hypoxia-inducible factor-1 via a non-proteolytic mechanism;Sakamoto;J. Biol. Chem.,2010

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Regulation of Mitochondrial Hydrogen Peroxide Availability by Protein S-glutathionylation;Cells;2022-12-27

2. Glutaredoxin 1 controls monocyte reprogramming during nutrient stress and protects mice against obesity and atherosclerosis in a sex-specific manner;Nature Communications;2022-02-10

3. Aggregated LDL turn human macrophages into foam cells and induce mitochondrial dysfunction without triggering oxidative or endoplasmic reticulum stress;PLOS ONE;2021-01-25

4. Redox RegulationviaGlutaredoxin-1 and ProteinS-Glutathionylation;Antioxidants & Redox Signaling;2020-04-01

5. Sexual dimorphism in glutathione metabolism and glutathione-dependent responses;Redox Biology;2020-04