Large-scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E ε2/ε3/ε4 genotype
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference5 articles.
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2. Apolipoprotein E alleles and risk of coronary disease: a meta-analysis;Wilson;Arterioscler Thromb Vasc Biol,1996
3. Utility of the predictors of coronary heart disease mortality in a longitudinal study of elderly Finnish men aged 65 to 84 years is dependent on context defined by APOE genotype and area of residence;Stengard;Clin Genet,1999
4. Apolipoprotein E4 and coronary heart disease in middle-aged men who smoke: a prospective study;Humphries;Lancet,2001
5. Large-scale test of hypothesised associations between the angiotensin-converting-enzyme insertion/deletion polymorphism and myocardial infarction in about 5000 cases and 6000 controls;Keavney;Lancet,2000
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1. From degenerative disease to malignant tumors: Insight to the function of ApoE;Biomedicine & Pharmacotherapy;2023-02
2. Apolipoprotein E Gene Variants and Risk of Coronary Heart Disease: A Meta-Analysis;BioMed Research International;2016
3. Association between Apolipoprotein E polymorphism and myocardial infarction risk: A systematic review and meta‐analysis;FEBS Open Bio;2015-01
4. A systematic review and meta-analysis of 130,000 individuals shows smoking does not modify the association of APOE genotype on risk of coronary heart disease;Atherosclerosis;2014-11
5. Meta-Analysis of Apolipoprotein E Gene Polymorphism and Susceptibility of Myocardial Infarction;PLoS ONE;2014-08-11
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