A prospective mechanism and source of cholesterol uptake by Plasmodium falciparum-infected erythrocytes co-cultured with HepG2 cells-Reference-Cited by-同舟云学术

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A prospective mechanism and source of cholesterol uptake by Plasmodium falciparum-infected erythrocytes co-cultured with HepG2 cells

Published:2021-02 Issue: Volume:80 Page:102179
ISSN:1383-5769
Container-title:Parasitology International
language:en
Short-container-title:Parasitology International

Author:

Hayakawa Eri H.,Kato Hirotomo,Nardone Glenn A.,Usukura Jiro

Funder

Japan Society for the Promotion of Science

Division of Intramural Research, National Institute of Allergy and Infectious Diseases

National Institutes of Health

Publisher

Elsevier BV

Subject

Infectious Diseases,Parasitology

Reference53 articles.

1. Effects of very small amounts of cholesterol on gel-phase phosphatidylcholine membranes;Subczynski;Biochim. Biophys. Acta,1986

2. Phosphatidylcholine-cholesterol interactions: bilayers of heteroacid lipids containing linoleate lose calorimetric transitions at low cholesterol concentration;Keough;Biochim. Biophys. Acta,1989

3. A calorimetric and spectroscopic comparison of the effects of ergosterol and cholesterol on the thermotropic phase behavior and organization of dipalmitoylphosphatidylcholine bilayer membranes;Mannock;Biochim. Biophys. Acta,2010

4. Cholesterol facilitates the native mechanism of Ca2+−triggered membrane fusion;Churchward;J. Cell Sci.,2005

5. A novel pathway for ca++ entry into Plasmodium falciparum-infected blood cells;Desai;Am. J. Trop. Med. Hyg.,1996

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4. Chemogenomic Profiling of a Plasmodium falciparum Transposon Mutant Library Reveals Shared Effects of Dihydroartemisinin and Bortezomib on Lipid Metabolism and Exported Proteins;Microbiology Spectrum;2023-06-15

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