Sheep models of polycystic ovary syndrome phenotype
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference148 articles.
1. Developmental origin of polycystic ovary syndrome – a hypothesis;Abbott;J. Endocrinol.,2002
2. Animal models and fetal programming of PCOS;Abbott,2006
3. Infants of women with polycystic ovary syndrome have lower cord blood androstenedione and estradiol levels;Anderson;J. Clin. Endocrinol. Metab.,2010
4. Levels of testosterone, androstenedione, estrone and estradiol-17 beta in the testes of fetal sheep;Attal;Endocrinology,1969
5. Positions statement: criteria for defining polycystic ovary syndrome as a predominantly hyperandrogenic syndrome: an Androgen Excess Society guideline;Azziz;J. Clin. Endocrinol. Metab.,2006
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1. Reproductive neuroendocrine defects programmed by prenatal testosterone treatment between gestational days 60–90 are amplified by postnatal obesity in sheep;Frontiers in Physiology;2024-08-02
2. Early- to mid-gestational testosterone excess leads to adverse cardiac outcomes in postpartum sheep;American Journal of Physiology-Heart and Circulatory Physiology;2024-08-01
3. Glutathione peroxidase (GPX1) - Selenocysteine metabolism preserves the follicular fluid's (FF) redox homeostasis via IGF-1- NMD cascade in follicular ovarian cysts (FOCs);Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-08
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