Methylation of the p16Ink4a tumor suppressor gene 5′-CpG island in breast cancer-Reference-Cited by-同舟云学术

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Methylation of the p16Ink4a tumor suppressor gene 5′-CpG island in breast cancer

Published:2001-02 Issue:1 Volume:163 Page:59-69
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Nielsen N.H.,Roos G.,Emdin S.O.,Landberg G.

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference40 articles.

1. DNA methylation errors in cancer;Jones;Cancer Res.,1996

2. Alterations in DNA methylation: a fundamental aspect of of neoplasia;Baylin;Adv. Cancer Res.,1998

3. Hypermethylation can selectively silence individual p16Ink4a alleles in neoplasia;Myöhänen;Cancer Res.,1998

4. Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complex;Nan;Nature,1998

5. Methylated DNA and MeCP2 recruit histone deacetylase to repress transcription;Jones;Nat. Genet.,1998

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2. Evaluation of rs10811661 polymorphism in CDKN2A / B in colon and gastric cancer;BMC Cancer;2023-10-16

3. Epigenetic Silencing of p16INK4a gene in Sporadic Breast Cancer;Indian Journal of Surgical Oncology;2023-06-20

4. Evaluation of rs10811661 polymorphism in CDKN2A / B in colon and gastric cancer;2023-03-13

5. Non-CpG methylation—a key epigenetic modification in cancer;Briefings in Functional Genomics;2021-07-27

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