Enhanced insulin-induced mitogenesis and mitogen-activated protein kinase activities in mutant insulin receptors with substitution of two COOH-terminal tyrosine autophosphorylation sites by phenylalanine.-Reference-Cited by-同舟云学术

Enhanced insulin-induced mitogenesis and mitogen-activated protein kinase activities in mutant insulin receptors with substitution of two COOH-terminal tyrosine autophosphorylation sites by phenylalanine.

Published:1992-06 Issue:18 Volume:267 Page:12788-12796
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Ando A,Momomura K,Tobe K,Yamamoto-Honda R,Sakura H,Tamori Y,Kaburagi Y,Koshio O,Akanuma Y,Yazaki Y

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference47 articles.

1. Multiple components in an epidermal growth factor-stimulated protein kinase cascade

2. An Insulin-Stimulated Protein Kinase Similar to Yeast Kinases Involved in Cell Cycle Control

3. ERKs: A family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF

4. Human insulin receptors mutated at the ATP-binding site lack protein tyrosine kinase activity and fail to mediate postreceptor effects of insulin.

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