Missense variant in insulin receptor (Y1355H) segregates in family with fatty liver disease-Reference-Cited by-同舟云学术

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Missense variant in insulin receptor (Y1355H) segregates in family with fatty liver disease

Published:2021-11 Issue: Volume:53 Page:101299
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Luo Fei^ORCID,Xing Chao^ORCID,Asrani Sumeet K.^ORCID,Li Shili,Liang Guosheng,Hobbs Helen H.^ORCID,Cohen Jonathan C.

Funder

National Institutes of Health

Howard Hughes Medical Institute

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference25 articles.

1. Phosphorylation activates the insulin receptor tyrosine protein kinase;Rosen;Proceedings of the National Academy of Sciences of the U S A,1983

2. Identification of insulin receptor tyrosine residues autophosphorylated in vitro;Tornqvist;Journal of Biological Chemistry,1987

3. Mechanisms of insulin action and insulin resistance;Petersen;Physiological Reviews,2018

4. The role of insulin receptor autophosphorylation in signal transduction;Murakami;Journal of Biological Chemistry,1991

5. Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucose;Ellis;Cell,1986

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Remnant cholesterol has a non-linear association with non-alcoholic fatty liver disease;Diabetes Research and Clinical Practice;2023-07

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