Traveling Bax and Forth from Mitochondria to Control Apoptosis
Author:
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference10 articles.
1. BCL-2, BCL-XL Sequester BH3 Domain-Only Molecules Preventing BAX- and BAK-Mediated Mitochondrial Apoptosis
2. Cell Death
3. Bcl-xL Retrotranslocates Bax from the Mitochondria into the Cytosol
4. The Soluble Form of Bax Regulates Mitochondrial Fusion via MFN2 Homotypic Complexes
5. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics
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