Deletion of DDB1 in Mouse Brain and Lens Leads to p53-Dependent Elimination of Proliferating Cells

Author:

Cang Yong,Zhang Jianxuan,Nicholas Sally A.,Bastien Jayson,Li Baojie,Zhou Pengbo,Goff Stephen P.

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference36 articles.

1. PCNA functions as a molecular platform to trigger Cdt1 destruction and prevent re-replication;Arias;Nat. Cell Biol.,2006

2. The ocular lens epithelium;Bhat;Biosci. Rep.,2001

3. Ddb1 is required for the proteolysis of the Schizosaccharomyces pombe replication inhibitor Spd1 during S phase and after DNA damage;Bondar;J. Biol. Chem.,2004

4. Cul4A and DDB1 associate with Skp2 to target p27Kip1 for proteolysis involving the COP9 signalosome;Bondar;Mol. Cell. Biol.,2006

5. Nucleotide excision repair syndromes: xeroderma pigmentosum, Cockayne syndrome, and trichothiodystrophy;Bootsma,2002

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