Low susceptibility of NC/Nga mice to tumor necrosis factor-α-mediated lethality and hepatocellular damage with d-galactosamine sensitization

Author:

Koide Naoki,Morikawa Akiko,Naiki Yoshikazu,Tumurkhuu Gantsetseg,Yoshida Tomoaki,Ikeda Hiroshi,Yokochi Takashi

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference34 articles.

1. Development of atopic dermatitis-like skin lesions with IgE hyperproduction in NC/Nga mice;Matsuda;Int. Immunol.,1997

2. IgE hyperproduction through enhanced tyrosine phosphorylation of Janus kinase 3 in NC/Nga mice, a model for human atopic dermatitis;Matsumoto;J. Immunol.,1999

3. Overproduction of Th2-specific chemokines in NC/Nga mice exhibiting atopic dermatitis-like lesions;Vestergaard;J. Clin. Invest.,1999

4. The mechanism of a defective IFN-gamma response to bacterial toxins in an atopic dermatitis model, NC/Nga mice, and the therapeutic effect of IFN-gamma, IL-12, or IL-18 on dermatitis;Habu;J. Immunol.,2001

5. Galactosamine-induced sensitization to the lethal effects of endotoxin;Galanos;Proc. Natl. Acad. Sci. U. S. A.,1979

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