The Mechanism of a Defective IFN-γ Response to Bacterial Toxins in an Atopic Dermatitis Model, NC/Nga Mice, and the Therapeutic Effect of IFN-γ, IL-12, or IL-18 on Dermatitis-Reference-Cited by-同舟云学术

The Mechanism of a Defective IFN-γ Response to Bacterial Toxins in an Atopic Dermatitis Model, NC/Nga Mice, and the Therapeutic Effect of IFN-γ, IL-12, or IL-18 on Dermatitis

Published:2001-05-01 Issue:9 Volume:166 Page:5439-5447
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Habu Yoshiko,Seki Shuhji,Takayama Eiji,Ohkawa Takashi,Koike Yuji,Ami Katsunori,Majima Takashi,Hiraide Hoshio

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference43 articles.

1. Allergic Skin Disorders and Mastocytosis

2. Decreased interferon gamma and increased interleukin-4 production in atopic dermatitis promotes IgE synthesis

3. Predominant TH2-like Bronchoalveolar T-Lymphocyte Population in Atopic Asthma

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