Degradation of phosphatidylinositol-4,5-bisphosphate is insensitive to Ca2+ mobilization in stimulated platelets
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference27 articles.
1. Platelets in Biology and Pathology-2;Gerrard,1981
2. Comparison of metal-binding properties of trans-1,2-cyclohexanediol diphosphate and deacylated phosphoinositides
3. Inositol phospholipids and cell surface receptor function
4. The initial action of thrombin on platelets. Conversion of phosphatidylinositol to phosphatidic acid preceding the production of arachidonic acid.
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1. The role of intracellular calcium in A-23187 stimulated and β-adrenoceptor blocking drug treated blood platelets;Biochemical Pharmacology;1994-06
2. Further characterization of 5-HT- and 5-HT3 receptor agonists'-stimulated phosphoinositol phosphates accumulation;Brain Research;1993-07
3. Chlorpromazine increases the turnover of metabolically active phosphoinositides and elevates the steady-state level of phosphatidylinositol-4-phosphate in human platelets;Biochemical Pharmacology;1992-11
4. Specific binding of tritium-labeled inositol 1,4,5-trisphosphate to human platelet membranes: Ionic and GTP regulation;Biochimica et Biophysica Acta (BBA) - Biomembranes;1991-05
5. Thrombin stimulates the production of phosphatidylinositol 3,4-bisphosphate in human erythroleukemia cells;Archives of Biochemistry and Biophysics;1991-05
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