Impaired cross-activation of β3 integrin and VEGFR-2 on endothelial progenitor cells with aging decreases angiogenesis in response to hypoxia-Reference-Cited by-同舟云学术

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Impaired cross-activation of β3 integrin and VEGFR-2 on endothelial progenitor cells with aging decreases angiogenesis in response to hypoxia

Published:2013-10 Issue:3 Volume:168 Page:2167-2176
ISSN:0167-5273
Container-title:International Journal of Cardiology
language:en
Short-container-title:International Journal of Cardiology

Author:

Di Qun,Cheng Zeen,Kim Weon,Liu Zexuan,Song Hui,Li Xiang,Nan Yongshan,Wang Chengya,Cheng Xianwu

Funder

National Natural Science Foundation of China

Bio & Medical Technology Development Program of the National Research Foundation (NRF)

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference30 articles.

1. Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: part I: aging arteries: a “set up” for vascular disease;Lakatta;Circulation,2003

2. Age decreases endothelial progenitor cell recruitment through decreases in hypoxia-inducible factor 1alpha stabilization during ischemia;Chang;Circulation,2007

3. Exercise training stimulates ischemia-induced neovascularization via phosphatidylinositol 3-kinase/Akt-dependent hypoxia-induced factor-1 alpha reactivation in mice of advanced age;Cheng;Circulation,2010

4. Inhibition of prolyl hydroxylase domain proteins promotes therapeutic revascularization;Loinard;Circulation,2009

5. The angiogenic response is dictated by beta3 integrin on bone marrow-derived cells;Feng;J Cell Biol,2008

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